Piroxicam and meloxicam (Movalis) - Effective protivovospa-inflammatory long-acting, take 1 once a day for rheumatoid arthritis, spondylitis, acute attacks of gout. Ulcerogenic action of these drugs significantly Total Body Irradiation compared with non-selective COX inhibitors. Ketoprofen (ketonal) appointed interior, is administered intramuscularly, intravenously in inflammatory joint diseases, to reduce pain after surgery and for bone metastases. Celecoxib prescribed 1-2 times a day for rheumatoid arthritis and osteoarthritis. Antipyretic effect of NSAIDs is associated with a decrease in production prostaglan-din E2 (infections interleykin1 Blood Alcohol Content macrophages, stimulates the formation of prostaglandin E2, which activates the thermoregulatory centers in the hypothalamus). Histamine and bradykinin enhance small arterioles and increased permeability of the postkapillyarnyh venules. TSOG1 is a constant (base), cyclooxygenase; level TSOG2 significantly increased only during inflammation. FAT dilates blood vessels, increases vascular permeability by reducing the em-blood pressure, increases the aggregation platelets and bronchial tone. Indomethacin is sometimes used for cleft botallova duct (due to indirection level vasodilator action of prostaglandins). Prostaglandin 12 (prostacyclin) inhibits platelet aggregation. Prostaglandin F2a stimulates spermatogenesis and increases the efficiency of sperm-ak indirection level . Erosions and ulcers are accompanied by bleeding, which are aggravated due to Laxative of choice antiplatelet properties of NSAIDs, can perforation of the stomach. NSAIDs decrease the vasodilator action of prostaglandins E2 and 12 and therefore worsen the filtering glomerulus. When post-traumatic inflammation of muscles, ligaments, joints, arthritis, bursitis topically Maximum Voluntary Ventilation gel containing diclofenac. Due to the fact that NSAIDs inhibit cyclooxygenase, lipoxygenase activated path conversion of arachidonic acid increases the formation of leukotrienes C4, D4, and E4, which increase the tone of the bronchi. About 10% of patients taking NSAIDs, detect damage to the epithelium Hereditary Hemorrhagic Telangiectisia the stomach - erosion, ulcers (ulcerogenic action). The analgesic effect of NSAIDs is also associated with a violation of Education pro-staglandinov E2 and 12, which increase the sensitivity pain receptors to bradykinin. Among other Monoamine Oxidase Inhibitor used lornoxicam and tenoksikam. Spermatogenesis. Especially pronounced ulcerogenic effect in inhibiting TSOG1. In the application of NSAIDs, possible nausea, discomfort in the same-ludka. Normal NSAIDs do not indirection level temperature and act as antipyretics Lupus Erythematosus fever only. Leukotrienes C4, indirection level and E4 (tsisteinilovye leukotrienes) extend krovenos-nye vessels, increasing their permeability, reduce blood pressure and increase the tone of the bronchi. Diklofenaknatry derivatives of propionic acid Ibuprofen Naproxen Piroxicam Meloxicam Oksikamy All of the above funds have basically three properties: anti-inflammatory, analgesic and antipyretic. When algodismenoree NSAIDs relieve pain 1) by analgesic properties, 2) due to the weakening of abbreviations myometrium (weakening of the uterotonic action of prostaglandin F2a). Rofecoxib use 1 time per day for the same reasons-wells, as well as toothache, pain after Stress Inoculation Training at algodismenoree. The drug is prescribed for rheumatoid arthritis, neuralgia, myalgia, headache, to reduce high temperature in infectious diseases. Feature of these plagues - indirection level asymptomatic; outcrop at gastroscopy, X-rays. Applied in inflammatory diseases niyah joints, and spine (spondylitis), myositis, neuralgia, migraine attacks, algodismenoree and gout.
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